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10 Foods that bring on the pain 6abc.com, PA - Nov 26, 2008 "Saturated fatty acids lead to the formation of arachidonic acid, which in turn converts to inflammatory substances called prostaglandins and leukotrienes, ...
Gene sources of human metabolic rates found iTWire, Australia - Nov 29, 2008 FADS1 is short for fatty acid desaturase 1. The protein encoded by this gene is a member of the fatty acid desaturase (FADS) gene family, whose enzymes ...
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Decoding holiday heartburn News Sentinel, IN - Nov 17, 2008 ?GERD is definitely on the rise,? Tiriveedhi said, due for the most part to obesity and the American diet of fried and high-fat foods and sugared drinks. ...
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Recent News and Articles on the Keywords: obesity + monitors + 0.25 Related to the article below (Last Update: 8/7/2008)
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[CITATION] Manuscript in press, Obesity CL Davis, J Tkacz, M Gregoski, CA Boyle, G … -
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Obesity: hypothalamus also monitors fatty acid levels and responds by controlling appetite
Researchers at the Albert Einstein College of Medicine have shown that reducing levels of fatty acids in the hypothalamus causes rats to overeat and become obese.
Their results suggest that restoring fatty-acid levels in the brain may be a promising way to treat obesity.
The study, published in the Nature Neuroscience, was led by Luciano Rossetti, director of the Diabetes Research Center at Einstein.
The brain's hypothalamus keeps track of the body's nutritional status by monitoring the blood levels of several different hormones and nutrients.
Taking this information into account, the hypothalamus regulates our energy intake and metabolism.
In a study published in Science, Rossetti and his colleagues showed how the hypothalamus monitors and regulates glucose levels in the body.
The present study shows that this brain region also monitors fatty acid levels and responds by controlling appetite.
The study focused on malonyl CoA, a molecule suspected of being one of the critical nutrients influencing hypothalamic regulation of eating behavior.
Previous studies had shown that hypothalamic levels of malonyl CoA increase markedly after meals and are suppressed by fasting.
The Einstein researchers wanted to know whether sustained suppression of this nutrient within the hypothalamus could result in obesity.
To find out, they piggybacked an enzyme known to degrade malonyl CoA onto an adeno-associated virus and injected the virus into the hypothalamus of rats. The injections caused a chronic decrease in malonyl CoA levels, which dramatically increased the rats' food intake and led to obesity that was maintained for at least four months.
" We showed in this study that disrupting malonyl-CoA levels in this region of the brain impairs the nutrient-sensing mechanism by which the hypothalamus modulates food intake to maintain normal weight," says Rossetti." Figuring out a way to re-adjust malonyl-CoA levels in the human hypothalamus could lead to innovative therapies not only to treat obesity but to help prevent diabetes and other consequences of being overweight."
